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Interleukin 22 Expands Transit-Amplifying Cells While Depleting Lgr5 Stem Cells via Inhibition of Wnt and Notch Signaling.

Cell Mol Gastroenterol Hepatol. 2019; 
ZhaJuan-Min，LiHua-Shan，LinQian，KuoWei-Ting，JiangZhi-Hui，TsaiPei-Yun，DingNing，WuJia，XuShao-Fang，WangYi-Tang，PanJian，ZhouXiu-Min，ChenKai，TaoMin，OdenwaldMatthew A，TamuraAtsushi，TsukitaSachiko，TurnerJerrold R，HeWe
Products/Services Used Details Operation
… mL). Blue symbols indicate enteroids cultured without IL22. Sources of IL22, from left to right, were GenScript Z03350, GenScript Z02197, R&D 582ML-010, R&D 582ML-010/CF, and PeproTech 201-22 (gold symbols). Each …

摘要

Epithelial regeneration is essential for homeostasis and repair of the mucosal barrier. In the context of infectious and immune-mediated intestinal disease， interleukin (IL) 22 is thought to augment these processes. We sought to define the mechanisms by which IL22 promotes mucosal healing.

关键词

Enteroid，Interleukin 22，Intestinal Stem Cell，Regeneration，Transit-Amplifying Cell，claudin-2，notch，tight junction